Project Details:

Rhg1, cqSCN Loci and Epigenetic Determinants of Resistance to Soybean Cyst Nematode (1920-172-0122-B)

Parent Project: Improved Resistance to Soybean Cyst Nematode via Rhg1 and Other Soybean Loci (Year 1 of 1420-532-5643)
Checkoff Organization:United Soybean Board
Categories:Nematodes, Breeding & genetics, Environmental stress
Organization Project Code:1920-172-0122-B
Project Year:2019
Lead Principal Investigator:Andrew Bent (University of Wisconsin)
Co-Principal Investigators:
Brian Diers (University of Illinois at Urbana-Champaign)
Matthew Hudson (University of Illinois at Urbana-Champaign)
Keywords: soybean cyst nematode, SCN, genetic resistance

Contributing Organizations

Funding Institutions

Information and Results

Comprehensive project details are posted online for three-years only, and final reports indefinitely. For more information on this project please contact this state soybean organization.

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Final Project Results

Updated December 10, 2019:
The research team made progress on multiple fronts in our identification and manipulation of plant traits that control soybean resistance to soybean cyst nematode.
Highlights include:
• Soybean germplasm development and characterization for both naturally occurring and transgenically developed new versions of the Rhg1 locus, which plays a major role in SCN resistance.
• Two scientific papers published in peer-reviewed journals, one reporting early gene expression responses of soybean to soybean cyst nematode HG type 0, and the other reporting the unexpected and usefully predictive presence of a "copia retrotransposon" (mobile DNA element) within one of the Peking-type Rhg1 genes.
• Two additional scientific manuscripts submitted to peer-reviewed journals, reporting that the gamma-SNAP encoded at cqSCN-006 contributes to SCN resistance, and reporting syntaxins that contribute to SCN resistance.
• Additional proteins identified by the research team that are contributors to SCN resistance.
• Achievement of new focus on a smaller set of candidate genes that are the likely cause of the SCN resistance trait conferred by the cqSCN-007 quantitative trait locus.
• Development and initial use of Rhg1 locus DNA methylation assays to assess the potential contribution of differential Rhg1 locus methylation to SCN resistance.

Project Years